Bee research, human sweet perception, human pheromones, and metabolic disorders
Posted on June 30, 2012 by James Kohl.
“Bee research sheds light on human sweet perception, metabolic disorders.” June 29th, 2012.
The epigenetic effects of nutrient chemicals, like glucose, and their metabolism to pheromones sheds light on how what is known about the honeybee model organism explains metabolic disorders and other disorders in humans. For example, I use the honeybee model organism to detail the molecular biology of brain development in mammals.
Nutrient chemicals and pheromones are essential to brain development in the honeybee, in mammals, and in us. A diet-reponsive neurogenic niche links nutrient chemical intake to receptor-mediated brain development in mammals. Glucose regulates the hormone secreting nerve cells in this niche, which links it and other nutrient chemicals to levels of luteinizing hormone (LH) and brain development. The same neurogenic niche links mammalian pheromones to LH. Thus, the diet-responsive neurogenic niche appears to also respond to pheromones that regulate brain development.
There are many human pheromone deniers, evolutionary theorists, and psychotherapists who think our brain development and behavior is not substantially altered by pheromones. They need to start thinking clearly. Denying the role of human pheromones in the context of brain development and behavior is like denying the role of food odors in brain development and behavior in all species from insects to other mammals and to us.
The same pathway is involved, and I’ve detailed it in Human pheromones and food odors: epigenetic influences on the socioaffective nature of evolved behaviors.
Retired medical laboratory scientist
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