Natural Selection and the Complexity of the Genome
Posted on September 16, 2012 by James Kohl.
Natural Selection and the Complexity of the Genome by James V. Kohl
In the Genetic Predispositions threads duplicated in Psychiatry-research and Evolutionary-psychology, there may be a few people who have grasped the concept of adaptive evolution, which requires selection for nutrient chemicals and species-specific mixtures of chemicals called pheromones in species from microbes to man. One participant suggested that any new chemical activity in the cell must trigger a fairly powerful selective force.
Nutrient chemicals and pheromones cause receptor-mediated chemical activity in the cell that triggers natural selection, which is a powerful selective force.
Last month, another participant who has repeatedly denigrated my published work, said in a different forum: “... I envision some unconditioned stimuli to have been such a prevalent and regular feature of past environments that it makes sense to bypass the normal learning mechanism and encode the process in the DNA.”
Nutrient chemicals and pheromones are prevalent unconditioned stimuli and they are also regular features of all past environments that encode normal receptor-mediated learning mechanisms in the cell via the chemical activity that causes natural selection, which is a powerful selective force.
Unfortunately, the moderator of both the psychiatry-research and evolutionary-psychology group misunderstood my conceptualization of how organisms are epigenetically fed the nutrient chemicals that enable gene duplication as a mechanism of genomic adaptation to a changing environment. He has not yet responded to my reply that “epigenetically fed” does not mean that DNA from one species becomes functional as DNA in another. Instead, receptor-mediated ingestion of the DNA of a heterospecific becomes the nutrient chemical that enables gene duplication in conspecifics, which is required for adaptive evolution via ecological, social, neurogenic, and socio-cognitive niche construction in species from microbes to man (Kohl 2012).
As an alternative to my model, one of the people involved in presenting the ENCODE data has said there are “random models” that may explain adaptive evolution across species. Does anyone know what he’s talking about? What “random model” helps to explain the species diversity that results from nutrient acquisition and the metabolism of nutrient chemicals to pheromones, which control reproduction in all species? If there is a “random model” that has any explanatory power not why haven’t the details of the model been offered for consideration. The “random models” proposal makes me think we will continue to be misled about the requirements for selection in the context of epigenetically altered cause and effect that involves the complexity of the genome and natural selection in all species?
Retired medical laboratory scientist
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