Parallel molecular evolution 2

October 10, 2012 | James Kohl

Mutations in the neverland Gene Turned Drosophila pacheainto an Obligate Specialist Species

  • Michael Lang,
  • st al with Virginie Orgogozo

Science 28 September 2012: 1658-1661. [DOI:10.1126/science.1224829]

My comment: Nutrient chemical-dependent epigenetic effects that lead to loss of enzymatic activities, which here are reported to be frequent in evolution, are not random. The metabolism of the nutrient chemicals to pheromones that control reproduction is genetically predisposed (e.g., not random). However, the nutrient chemical-dependent speciation that is reportedly due to loss of enzymatic function is also reported in the context of mutations that caused speciation. How does a mutation cause nutrient chemical-dependent / pheromone-controlled obligate specialist speciation (or any other type of nutrient chemical-dependent speciation)?

Is it commonly known that flies reproduce at the same location that they find nutrient chemicals that metabolize to the pheromones that control their species-specific reproduction, or has that concept escaped the notice of evolutionary theorists during the past two centuries of disgust, which is sometimes associated with flies landing on their food?

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James Vaughn Kohl

James Vaughn Kohl

James Vaughn Kohl was the first to accurately conceptualize human pheromones, and began presenting his findings to the scientific community in 1992. He continues to present to, and publish for, diverse scientific and lay audiences, while constantly monitoring the scientific presses for new information that is relevant to the development of his initial and ongoing conceptualization of human pheromones.