Is the model for the development of food preferences and development of sexual preferences from species of microbes to man supported equally well by the same data?
the only thing wrong with mathematical models of cause and effect is that they attribute indirect genetic effects, direct genetic effects, and affects on behavior to something unknown
A systems biology approach to chronic fatigue that incorporates neuroscientifically established fact is obviously required.
A computational model? It is model organisms that explain the epigenetic effects of sensory input on the development of behavior and species survival.
A detailed model exemplifies the effects of olfactory/pheromonal conditioning, which alters genetically predisposed, nutrient chemical-dependent, hormone-driven mammalian behavior and choices for pheromones that control reproduction via their effects on luteinizing hormone.
On November 26, 2012 Robert Karl Stonjek announced to the members of the psychiatry research group: Dear Members, there will be no more discussion of JVKs ‘model’.