“… the “mutational vicinity” is probably found in the molecular vicinity of a “metabolic niche”.
Shall we attribute to him the inability to see that metabolism and epigenetics converge with food odors and the metabolism of nutrient chemicals to pheromones that act on precisely the same molecular mechanisms of adaptive evolution found in species from microbes to man?
I think what they’ve just shown us exemplifies the molecular biology of epigenetically driven adaptive evolution.
The authors are among many, who seem to have missed a likely epigenetic link from maternal and/or acquired ferritin deficiency to thyroxine transport, brain development, and behavior.
the logic of gene regulatory networks in adaptive evolution does not skip any species.
Across species comparisons of epigenetic effects on genetically predisposed nutrient-dependent and hormone-driven invertebrate and vertebrate social and sexual behavior indicate that human pheromones also alter the development of the brain and behavior via the same molecular mechanisms.
The epigenetic effects of nutrient chemicals on gene duplication and species diversity and the ability to metabolize nutrient chemicals is controlled by the metabolism of the nutrient chemicals to pheromones that epigenetically control reproduction in all species
We incorporated what is currently known about the ability of chemical signals to condition behavior. This conditioning occurs via epigenetic effects that calibrate and standardize the molecular biology of intracellular signal transduction and stochastic gene expression
The epigenetic effects are on interactions among all the other neuronal systems and hormone secretion that interact with the GnRH neuronal system, which is central to all developmental differences in the brain of males and females.