The specialist-generalist dilemma: Is evolutionary theory dead?
April 30, 2014 | James Kohl
Science and the Humanities (1971)
“Schrödinger closes his discussion of the specialist-generalist dilemma with the following words: [(147), p. 112]: Never lose sight of the role that your particular subject has within the great performance of the tragi-comedy of human life; keep in touch with life – not so much with practical life as with the ideal background of life, which is ever so much more important; and, Keep life in touch with you. If you cannot – in the long run – tell everyone what you have been doing, your doing has been worthless.”
As a generalist/medical laboratory scientist, I’ve been detailing some of the facts that help to link science and the humanities:
1) Life is nutrient-dependent.
2) Darwin’s ‘conditions of life” are nutrient-dependent.
3) Species-specific pheromones control the physiology of reproduction and nutrient-dependent species diversity.
4) Mutations do not!
5) Mutation-initiated natural selection is pseudoscientific nonsense.
6) Mutation-driven evolution also is pseudoscientific nonsense.
7) Specialization has resulted in generations of social scientists who have touted pseudoscientific nonsense.
Schrödinger’s cat is not dead. Pandora let it out of the box and evolutionary theorists pretended that issues of physics, chemistry, and conserved molecular mechanisms could best be explained away by a theory that: “Random mutations are the substrates on which directional natural selection acts.” Stories continue to be told that incorporate that widely popular theory.
See the latest pseudoscientific nonsense for example: Repair of naturally occurring mismatches can induce mutations in flanking DNA
“… the normal cycling of the epigenetic methyl-C mark could contribute to the high mutation rate of regulatory sequences thought to contribute both disease processes (Maurano et al., 2012) and evolutionary novelty (Wittkopp and Kalay, 2012).”
What these authors have done is link their thoughts about nutritional epigenetics and nutrient-dependent pheromone-controlled DNA methylation, which is obviously the cause of species diversity, to mutations that somehow cause disease and evolution.